Populus albus

Populus Albus

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Salicaceae

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Tree

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White poplar

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The Od Force of the closed buds blows branch crust and blows sheet buds is used to prepare this component.This Od Force in question activates the deactivated cell to cell negatively charged electromagnetic pathways networks/circuits as caused by the materials that have functioned to deal with the anodyne, anti-inflammatory, antiseptic, astringent, diuretic and tonic. It is used to cancel the results obtained from the material treatments of rheumatism, arthritis, gout, lower back pains, urinary complaints, digestive and liver disorders, debility, anorexia, also to reduce fevers and relieve the pain of menstrual cramps. It also reestablishes the activities of the deactivated cell to cell negatively charged electromagnetic pathways networks/circuits in the external material or chemical treatments in the chilblains, haemorrhoids, infected wounds and sprains. The results in the material or chemical treatments of caries of teeth and bones are reversed herewith as it activates the concerning electromagnetic fields to reproduce the corresponding constructive Od Force.

The material/chemical functions reputed for possessing pain-alleviating and anti-inflammatory properties are cancelled herewith. The consequences from the use of the material in the treatments of the arthritic and rheumatic pains and aches help in detaching the concerned Od Force that had constituted the lost one(s) is also under this Od Force to wash out. Moreover the material or chemical diversion/shifts from the material effects in reducing fever and fever particularly when the condition is related to rheumatic arthritis.

It withdraws the material stimulation electromagnetic recordings of works as an energizer while treating anorexia as well as other different conditions related to debility. Similarly it deletes material or chemical possessions over the antiseptic and astringent attributes that make it effective while treating diarrhea as well as symptoms of irritable bowel syndrome ( IBS), even conditions somewhat below old diarrhea. The material or chemical shifts caused from the process of promoting digestion and augmenting the functions of liver and stomach in case of loss of appetite are erased herewith.

Other material or chemical benefits including its usefulness in treating infections of the urinary tract and its effectiveness in feverish cold as well as infections causing cystitis are dealt firmly to rewind the process. The other uses of the material to include the treatments of the weakness of the female organs, urine incontinence, dysmenorrhea (painful menstruation) as well as discharge of mucus from the bladder are also taken into considerations to root out the caused Od Force deficiency.
The dangerous deactivation of the cell to cell negatively charged electromagnetic pathways network(s)/circuit(s) along with destruction of its related material after the use of material blood thinner is withdrawn herewith. Augmented risk of bleeding in the stomach is erased herewith.
The chemical or material diversions caused in the material or chemical treatment of caries of teeth and bones is removed herewith. It withdraws the skin reactions, vomiting, liver toxicity, allergic affections, nausea, heartburn. It erases the diuretic footprints of gonorrhea. Material’s depurative functions are withdrawn herewith. It is effective in burns and cold. Reducing of pain and inflammation that are associated with sore throats, arthritis, colds, headaches, rheumatism, fevers, and strained or aching muscles are subjected to the loss of the Od Force from the human system. The ease of symptoms of cystitis and enlarged prostate, as received from the material or chemical diuretic treatment are erased hereby to lift the focus of diversion caused by the chemical or material treatments. The material or chemical activities in the strong astringent and antibacterial action relieves many skin conditions, including burns, cuts, wounds, abrasions and that material or chemical activities may help loosen and expel mucus to ease coughs, congestion and respiratory infections. But all these are chemical or material diversion or shifts. These shifts hide those sufferings through causation of some new focus denoting the sum of the loss of the Od Force that had been caused earlier to cause those sufferings as are being treated now plus the loss by the treatment as done now and as a result the pain impulses may be blocked in brain, fever control thermostat may be changed in brain. It is oxidant in performing its duty.
Threatening sufferings appeared after the material or chemical treatments of hazards of pregnancy, chronic disease of the gastrointestinal tract, such as stomach or duodenal ulcers, reflux esophagitis, ulcerative colitis, spastic colitis, diverticulosis or diverticulitis are lifted herewith.
Its functions are notable where the chemical or material diversions or shifts cause from arthritis and rheumatism where there is much pain and swelling. Its means this Od Force is most effective when the diversions or shifts have been caused from a broad therapeutic area which is different when the therapeutic zone is small or simply other. If these diversions or shifts are caused from the flare-up of rheumatoid arthritis it is most applicable. A chemical or material that functions as a cholagogue, stimulates the digestion and specially stomach and liver function particularly where there is loss of appetite. The cellular damages done by the infections causing feverish cold and infections such as cystitis are withdrawn herewith. The material or chemical astringency that has been used to treat diarrhea/dysentery is rewind herewith with the lift of the astringency as the applied Od Force generates the loss of the material or chemical caused loss during the process of astringency.
Such diversions or shifts from the material or chemical treatments of the pathologies like intermittent fever, protracted fevers, prostatic hypertrophy and general debility are subjected to easily withdrawing.

The diversion of chemical shifts from the sufferings of tinnitus ringing of the ears, Itchy skin, hives or rashes, asthma and other breathing difficulties, angioedema, headaches, swelling of hands, feet, eyelids, face and/or lips, bed wetting or urgency to pass water, persistent cough, changes in skin color/skin discoloration, fatigue, sore, itchy, puffy or burning eyes, sinusitis/nasal polyps, diarrhea, hyperactivity, memory loss and poor concentration, depression, pseudoanaphylaxis are withdrawn herewith.

It works through several different pathways. It rewinds the material procedures that produce its anti-inflammatory effects via suppressing the activity of cyclooxygenase (COX), an enzyme that is responsible for the production of pro-inflammatory mediators such as the prostaglandins. It does this not by direct exhibition of the inhibition of COX like most other non-steroidal anti-inflammatory material but instead by expression of the suppression of the enzyme. It deactivates adenosine monophosphate-activated protein kinase (AMPK) and this action plays a role in withdrawing the anticancer effects of the material. The material or chemical antidiabetic effects are likely mediated by AMPK activation primarily through allosteric conformational change that increases levels of phosphorylation. This process is reversed herewith.

It also challenges the uncoupling oxidative phosphorylation, which leads to increased ADP: ATP and AMP:ATP ratios in the cell to reverse the same in opposite direction. As a consequence, this Od Force alters the alter AMPK activity and work done as an anti-diabetic by altering the energy status of the cell is cancelled herewith. AMPK knock-out displayed as an anti-diabetic effect, demonstrating at least one additional is cancelled herewith.

It affects the regulated c-Myc level at both transcriptional and post-transcription levels. Inhibition of c-Myc may be an important pathway by which the selected material or chemical exerts an anti-cancer effect, decreasing the occurrence of cancer in epithelial tissues at the cost of the material or chemical diversion or shifts is cancelled herewith to exhibits the rewinding paths in this respect.

It works to reverse keratolytic, comedolytic and bacteriostatic materiality that cause the cells of the epidermis to shed more readily, opening clogged pores and neutralizing bacteria within, preventing pores from clogging up again and allowing room for new cell growth. The diversion or shifts from these chemical or material activities are washed herewith.

The ototoxic effect caused at the cost of inhibiting prestin is withdrawn herewith. The materially or chemically induced transient hearing loss in zinc-deficiency is withdrawing here. In its functions the Od Force in question what does in induced hearing loss in zinc-deficiency, an injection of zinc helps get back the hearing that had been lost. But this recovery caused by the zinc injection takes place at the cost of loss of the Od Force as zinc causes chemical or material reactions with the cellular or chemical matter of the human system. As a result the human system here loses further Od Force or the electromagnetic force that had made intact the lost matter in the cell or cells. So due to loss of this Od Force a new focus of deficiency is created which signifies more troublesome sufferings of the patient.

Late pregnancy associated with bleeding, especially intracranial bleeding is withdrawn herewith. It cancels the material or chemical teratogenic potential, if any, how much low it may be. It withdraws the state of metabolic acidosis with compensatory respiratory alkalosis to reject morbidity and mortality though a process of chemical rewinding as the Od Force questioned here functions. In case of Reye's Syndrome its use could not be overlooked.

Cancelling the loss of the Od Force that was caused to perform the material or chemical action of hyper pigmentation over the skin without a broad spectrum sun block is withdrawn herewith.

Inhibition of the action of two enzymes called cyclooxgenase is hereby exhibited. Normally these enzymes convert an essential fatty acid called arachidonic acid into prostaglandins - hormone-like chemicals responsible for pain and inflammation - and leukotrienes. In doing so conversion some chemical reactions take place in which the Od Force of the cell as well as some cell matters is exhausted. So that a new focus is caused from this loss as well as from the loss of the Od Force due to previous inhibition. When the production of prostaglandins is made blocked, more leukotrienes are produced instead, becoming a problem for people who are sensitive to leukotrienes.  Leukotrienes are involved in a wide range of inflammatory conditions. The Od Force in question here meets up all the loss here by vibrating the respective cell to cell negatively charged electromagnetic circuit(s).

Other material or chemical diversion or shifts caused from the actions including fever reduction, general anti-inflammatory effect, increased volume and concentration of bile and rubefacient actions from external use is erased herewith. The shifts or diversions resulted from anti-inflammatory effect of anti-prostaglandin and bradykinin actions are rewind herewith. The diversions or shifts from the material or chemical activities that function as anti-septic, anthelminitic and caustic in the human material/chemical system are withdrawn herewith.

The materials of glycosides consist of secondary metabolites. These metabolites are tied to monosaccharide or an oligosaccharide or to uronic acid. This part of monosaccharide or uronic acid is termed as glycone and whatever the other molecule this glycon is attached to is named as aglycon. Flavonoids, frequently occurring as glycosides, are aglycones and whatever the saccharides these are attached to are the glycones. The glycone and aglycone are attached via a glycosidic linkage. Because of the diversity of aglycones and glycosides, these compounds do not share a common synthesis pathway. The process that forms the glycosidic linkage is termed as glycosylation and this glycosylation is caused through the accomplishment of specialized enzymes that use a uridine diphosphosugar as the glycone source. The addition of the glycone usually renders the total glycoside more polar and thus more water soluble than the isolated aglycone. The glycosidic linkage is relatively fragile. Several existing enzymes can break this link, including beta-glu-cosidase and beta-galactosidase. In order to maintain this fragile status those enzymes may or may not exist in the gastrointestinal lumen but they almost always occur in liver. The pharmacokinetics of glycosides is complex but central to an understanding of a built-in delayed release system, in which the hydrophilic glycoside delivers a hydrophobic aglycone to the large intestines. Most intact glycosides generally pass through the stomach and small intestines unchanged. However, some glycosides are absorbed, apparently by the intestinal epithelial intestinal Na+/ glucose monosaccharide cotransporter, SGLT-1(Sodium-glucose linked transporter-1). It may be discussed here that Sodium-dependent glucose co-transporters or sodium-glucose linked transporter, SGLT) are a family of glucose transporter found in the intestinal mucosa (enterocytes) of the small  intestine (SGLT1) and the proximal tubule of the nephron (SGLT2 in PCT and SGLT1 in PST). It may further be mentioned here that the proximal tubule is the portion of the duct system of the nephron of the kidney which leads from Bowman's capsule to the loop of Henle. It is conventionally divided into the proximal convoluted tubule (PCT) and the proximal straight tubule (PST). In the colon, the glycosidic bond is hydrolyzed by enzymes had by the gut flora. Some aglycones released by this process may also undergo enzymatic modification by gut bacteria. Aglycones are then absorbed via the large intestine, undergo first-pass metabolism in the liver, and circulate to varying degrees within the body. Glycoside aglycones released by the action of gut flora are more potent antineoplastic material or chemical. In withdrawing the above chemicals or material diversion or shifts this Od Force rewind the process. The loss of the Od Force of the cell to cell negatively charged electromagnetic circuits/network as caused by the chemical activities by the material antineoplastic is erased herewith. The process starts to be reversed as soon as the Od Force in question starts to function.

On the other hand, all cardiac glycosides inhibit Na⁺/K⁺-ATPase pumps throughout the body. Because these pumps are most concentrated and critical in cardiac myocytes, they have their greatest effect on this tissue. When an action potential passes through a cardiac myocyte, the cardiac glycoside limits sodium outflow and potassium inflow from the cell. The linked Na⁺/Ca²⁺ pump thus does not have sufficient sodium available to move calcium out of the cell. The higher than normal intracellular calcium concentration means that during the next action, potential contractility is increased. This is a positive inotropic action. Through another consequence this material or chemical activity reduces heart rate via a negative chronotropic action. Materially or chemically the glycosides limit conduction velocity in the atrioventricular node through the action of negative dromotropic. All of these effects, combined with a mild tendency to reduce tubular resorption of sodium in the kidneys and some moderate vaso-constrictive effects relieve the sufferings of congestive heart failure (CHF). They can also reduce some atrial arrhythmias. A lesser degree of hydroxylation correlates with rapid oral absorption and rapid renal excretion; greater hydroxylation slows oral absorption and is associated with biliary excretion, significant enterohepatic recirculation, and long half-lives.

The material or chemical diversion may focus in the zone of the cell to cell negatively charged electromagnetic circuit(s) that are allotted for the material or chemical diversions of cardiac glycosides to give the effects of cure to the patients.

The materials or chemicals of antibiotics may also unpredictably alter the effects of glycosides by changing the gut flora, thus altering enterohepatic recirculation. Any change in metabolism, such as fever, kidney failure, or intervening hyperthyroidism, can also dramatically alter the pharmacokinetics and thus the pharmacodynamics of these materials or chemicals. This Od Force is highly capable of intervening in this process of altered pharmacokinetics as well as pharmacodynamics to rewind the same to make free the suffering human system lifting from the deep den of further loss of the Od Force.

The material or chemical diversion or shifts may be to cause creation of new focus in the cell to cell negatively charged electromagnetic circuitry the flowing electromagnetic energy of which protects the same from the incidence of inhibition of the DNA polymerase from the inhibitory chemical reactions. This new focus may be seen in the zone that may undergo material or chemical inductions over water and electrolyte secretion as well as peristalsis including catharsis. The material or chemical diversion or shift may enter the circuit/sub-circuit that carries the electromagnetic force to keep intact the cell to cell framework that governs against the contraction of colon fairly strong and even painful griping, carminative or antispasmodic. It may even intrude in to induce atonic constitution as the colon adapts to the cathartic impulses. This diversion or shifts may jerk the cell to cell electromagnetic frameworks that are involved in leading against the diarrhea with fluid and electrolytes loss and ultimately, rhabdomyolysis, renal failure and other severe outcomes. The material or chemical diversion of cell to cell negatively charged electromagnetic frameworks may be of chemical reaction in the colon wall left with dark brown or black patches of pseudomelanosis coli is withdrawn or erased herewith. The shift or diversion may be focused in the inhibiting cell to cell negatively charged electromagnetic circuitry functions to cause excessive renal tubular cell proliferation, delay deterioration of patients in renal failure, modulated inflammation by partially inhibiting cyclooxygenase. The material or chemical diversion or shift may focus in the cell to cell negatively charged electromagnetic frameworks of iodine organification, promoting goiter and hypothyroidism. The circuitry of headache, bronchial constriction and weakness may also be affected. All these diversions or shifts mostly remain behind the curtain. But as this Od Force functions to delete all the noticeable diversions or sufferings the unnoticed loss of the Od Force the sufferings of which remain behind the curtain, are meet up herewith.