Vinca minor

Vinca minor

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Apocynaceae                                              

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Shrub

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Lessar periwinkle

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The Od Force of the whole plant with roots and fruits is used as one of the components in the preparation of the remedy. It is non-poisonous chemically.

The Od Force pacifies the stimulated blood circulation to the brain that has been made to enhance cognition in patients with dementia and enhance memory and learning in patients with degenerative and vascular dementia in the brain diseases. Dementia may be caused due to insufficient blood supply to the brain. The required blood circulation may be caused chemically but at the cost of further deficiency in the total already remained fund of the Od Force of the considered human system. This stimulation will naturally cause a new focus to show that the stimulated blood circulation has cured the diseases due to insufficient supply of blood like dementia.

The arteriosclerosis insufficient blood circulation will also create a new disease in the same way when that is ‘cured’ chemically in the same way presenting us a more grave new disease. And this Od Force will also do its best job to erase out the loss of the Od Force caused in the process of ‘curing’ the arteriosclerosis.

The Od Force in question here posses such quality energic vibrations that this Od Force salvages the human system from the fathomless severities of loss of the Od Forces caused by the chemically treated leukemia, lymphoma and other cancer. It is anti-astringent and withdraws the focus appeared due to the causation of healing outside the human physique. It withdraws the diversions caused by the chemically treated internal bleeding, heavy menstrual bleeding and even nose bleeding, bleeding haemorrhoids as if vasodilator. It withdraws the antispasmodic activities or established antispasmodic activities. It adds the hypotensive loss of the Od Force of the human system.

This Od Force is a Microtubule-constructive remedy. Different wave parts of this Od Force put itself differently to make free the Microtubule from its oppression done chemically during the cytotoxicity activities. Generally at the zone of low frequency that is, at low dilutions it withdraws the suppression or oppression done chemically on the Microtubule dynamics. The higher frequency of the higher dilutions vibrates the deactivated electromagnetic network field of the suffering human system to construct its microtubule polymer mass that is, the Od Force in question gradually lifts the imposed reducing activities that reduce the microtubule polymer mass. The Od Force in question here also applies itself against the playing activity that produces microtubule fragments by stimulating microtubule minus-end detachment from their organizing centers. The Od Force here cancels further the performances of enhancing microtubule detachment from spindle poles correlated best with cytotoxicity.

The spindle is a dynamic self-assembling machine that coordinates mitosis. The spindle’s function depends on its ability to organize microtubules into poles and maintain pole structure despite mechanical challenges and component turnover. It withdraws that force which disorganizes the activity that rapidly and robustly pulls severed microtubules and chromosomes poleward overpowering opposing forces and regaining the spindle architecture. The cytotoxicity imposes this disorganizing activity if it is in its capacity. It helps the NuMA and dynein/dynactin to regain their mediating duty towards the pole formation. Transport is powered by dynein pulling on the minus ends of severed microtubules. NuMA and dynein/dynactin are specifically enriched at new minus ends within seconds reanchoring minus ends to the spindle and delivering them to poles. This force on minus ends represents a newly uncovered chromosome transport mechanism that is independent of plus end forces at kinetochores and is well suited to robustly maintain the spindle mechanical integrity. The chemical reactions caused during the period of the cytotoxicity makes minus of that Od Force which organizes the above functions. And naturally the said Od Force escorts the deficient human system in the land of the plus of that Od Force that had been robbed during the time of the cytotoxicity.

Microtubules are required for the establishment of cell polarity, polarized migration of cells, intracellular vesicle transport, and chromosomal segregation in mitosis. Microtubules (MTs) are non-equilibrium polymers of α/β-tubulin heterodimers, in which GTP hydrolysis on the β-tubulin subunit occurs following assembly. Most microtubules are nucleated from organizing centers. The most prevalent microtubule behavior is dynamic instability, a process of slow plus end growth coupled with rapid depolymerization (“catastrophe”) and subsequent rescue. Although microtubule minus ends show dynamic instability, albeit at a lower rate than the plus ends show dynamic instability, the minus ends are usually capped and anchored at MT organizing centers and thus often do not participate in microtubule dynamics.

Maintaining a balance between dynamically unstable and stable microtubules is regulated in large part by proteins that bind either tubulin dimers or assembled microtubules. Proteins that bind tubulin dimers include stathmin, which sequesters tubulin and enhances MT dynamics by increasing catastrophe frequency, and collapsin response mediator protein (CRMP2), which increases MT growth rate by promoting addition of tubulin dimers onto microtubule plus ends. Other proteins that associate with assembled MTs include those that bundle MTs (e.g. MAP1c), those that stabilize MTs (e.g. tau), and those that maintain MTs in a dynamic state (MAP1b). A major signaling pathway that regulates MT dynamics involves GSK-3β, a kinase typically active under basal growth conditions but locally inactive in response to signals that enhance MT growth and dynamics.

In addition to the above factors, many MT motor proteins, and even non-motor proteins, aid in the dynamics of MTs. Proteins such as Xenopus microtubule associated protein 215 (XMAP215), promote MT assembly through binding to tubulin dimer to facilitate its incorporation in the growing plus end. XMAP215 also may compete with some of the MT plus end binding proteins (+TIPS), of which the end binding protein EB1 appears to be the master organizer. Complexes between the adenomateous polyposis coli (APC) protein and plus end binding proteins stabilize MTs by increasing the duration of the MT elongation phase. MT instability is promoted by several nonmotile kinesins from the kinesin-13 family. The mitotic centromere associated kinesin, MCAK, one of the most studied kinesin-13 family proteins, binds both plus and minus MT ends in vitro. The binding of MCAK to a MT end is thought to accelerate the transition to catastrophe by weakening the lateral interactions between the protofilaments

Tubulin undergoes several post-translational modifications such as acetylation, polyglutamylation, and poly-glycylation, which have been shown to alter the association with certain MT motors as well as other proteins that can affect MT stability and dynamics.

 When the concerned cytotoxicity causes the chemical reactions between the Microtubules and the chemical employed depending upon the situation the total phase may be affected selectively. Whatever be the dilution, whatever the part of the total reaction as stated above be affected during the establishing period of the cytotoxicity the concerned Od Force reverses the same to salvage the human system from the lacks of the Od Force as became recorded in the gene to its previous state.

Cancerous tumors are characterized by cell division, which is no longer controlled as it is in normal tissue.   "Normal" cells stop dividing when they come into contact with like cells, a mechanism known as contact inhibition.  Cancerous cells lose this ability.  Cancer cells no longer have the normal checks and balances in place that control and limit cell division.  The process of cell division, whether normal or cancerous cells, is through the cell cycle.  The cell cycle goes from the resting phase, through active growing phases, and then to mitosis (division).

The ability of chemotherapy to kill cancer cells depends on its ability to halt cell division.  Usually, the drugs work by damaging the RNA or DNA that tells the cell how to copy itself in division.  If the cells are unable to divide, they die.  The faster the cells are dividing, the more likely it is that chemotherapy will kill the cells, causing the tumor to shrink.  They also induce cell suicide (self-death or apoptosis).

Chemotherapy drugs that affect cells only when they are dividing are called cell-cycle specific.  Chemotherapy drugs that affect cells when they are at rest are called cell-cycle non-specific.  The scheduling of chemotherapy is set based on the type of cells, rate at which they divide, and the time at which a given drug is likely to be effective.  This is why chemotherapy is typically given in cycles.

Chemotherapy is most effective at killing cells that are rapidly dividing.  Unfortunately, chemotherapy does not know the difference between the cancerous cells and the normal cells. The "normal" cells will grow back and be healthy but in the meantime, side effects occur.  The "normal" cells most commonly affected by chemotherapy are the blood cells, the cells in the mouth, stomach and bowel, and the hair follicles; resulting in low blood counts, mouth sores, nausea, diarrhea, and/or hair loss.  Different drugs may affect different parts of the body.

transport is powered by dynein pulling on minus ends of severed microtubules. NuMA and dynein/dynactin are specifically enriched at new minus ends within seconds, reanchoring minus ends to the spindle and delivering them to poles. This force on minus ends represents a newly uncovered chromosome transport mechanism that is independent of plus end forces at kinetochores and is well suited to robustly maintain spindle mechanical integrity.

The spindle is a dynamic self-assembling machine that coordinates mitosis. The spindle’s function depends on its ability to organize microtubules into poles and maintain pole structure despite mechanical challenges and component turnover. Although we know that dynein and NuMA mediate pole formation, our understanding of the forces dynamically maintaining poles is limited: we do not know where and how quickly they act or their strength and structural impact. Using laser ablation to cut spindle microtubules, we identify a force that rapidly and robustly pulls severed microtubules and chromosomes poleward, overpowering opposing forces and repairing spindle architecture. Molecular imaging and biophysical analysis suggest that transport is powered by dynein pulling on minus ends of severed microtubules. NuMA and dynein/dynactin are specifically enriched at new minus ends within seconds, reanchoring minus ends to the spindle and delivering them to poles. This force on minus ends represents a newly uncovered chromosome transport mechanism that is independent of plus end forces at kinetochores and is well suited to robustly maintain spindle mechanical integrity.

Microtubules are required for the establishment of cell polarity, polarized migration of cells, intracellular vesicle transport, and chromosomal segregation in mitosis. Microtubules (MTs) are non-equilibrium polymers of α/β-tubulin heterodimers, in which GTP hydrolysis on the β-tubulin subunit occurs following assembly. Most microtubules are nucleated from organizing centers. The most prevalent microtubule behavior is dynamic instability, a process of slow plus end growth coupled with rapid depolymerization (“catastrophe”) and subsequent rescue. Although microtubule minus ends show dynamic instability, albeit at a lower rate than the plus ends show dynamic instability, the minus ends are usually capped and anchored at MT organizing centers and thus often do not participate in microtubule dynamics.

Maintaining a balance between dynamically unstable and stable microtubules is regulated in large part by proteins that bind either tubulin dimers or assembled microtubules. Proteins that bind tubulin dimers include stathmin, which sequesters tubulin and enhances MT dynamics by increasing catastrophe frequency, and collapsin response mediator protein (CRMP2), which increases MT growth rate by promoting addition of tubulin dimers onto microtubule plus ends. Other proteins that associate with assembled MTs include those that bundle MTs (e.g. MAP1c), those that stabilize MTs (e.g. tau), and those that maintain MTs in a dynamic state (MAP1b). A major signaling pathway that regulates MT dynamics involves GSK-3β, a kinase typically active under basal growth conditions but locally inactive in response to signals that enhance MT growth and dynamics.

In addition to the above factors, many MT motor proteins, and even non-motor proteins, aid in the dynamics of MTs. Proteins such as Xenopus microtubule associated protein 215 (XMAP215), promote MT assembly through binding to tubulin dimer to facilitate its incorporation in the growing plus end. XMAP215 also may compete with some of the MT plus end binding proteins (+TIPS), of which the end binding protein EB1 appears to be the master organizer. Complexes between the adenomateous polyposis coli (APC) protein and plus end binding proteins stabilize MTs by increasing the duration of the MT elongation phase. MT instability is promoted by several nonmotile kinesins from the kinesin-13 family. The mitotic centromere associated kinesin, MCAK, one of the most studied kinesin-13 family proteins, binds both plus and minus MT ends in vitro. The binding of MCAK to a MT end is thought to accelerate the transition to catastrophe by weakening the lateral interactions between the protofilaments

Tubulin undergoes several post-translational modifications such as acetylation, polyglutamylation, and poly-glycylation, which have been shown to alter the association with certain MT motors as well as other proteins that can affect MT stability and dynamics.

The spindle is a dynamic self-assembling machine that coordinates mitosis. The spindle’s function depends on its ability to organize microtubules into poles and maintain pole structure despite mechanical challenges and component turnover. Although we know that dynein and NuMA mediate pole formation, our understanding of the forces dynamically maintaining poles is limited: we do not know where and how quickly they act or their strength and structural impact. Using laser ablation to cut spindle microtubules, we identify a force that rapidly and robustly pulls severed microtubules and chromosomes poleward, overpowering opposing forces and repairing spindle architecture. Molecular imaging and biophysical analysis suggest that transport is powered by dynein pulling on minus ends of severed microtubules. NuMA and dynein/dynactin are specifically enriched at new minus ends within seconds, reanchoring minus ends to the spindle and delivering them to poles. This force on minus ends represents a newly uncovered chromosome transport mechanism that is independent of plus end forces at kinetochores and is well suited to robustly maintain spindle mechanical integrity.

Thuja occidentalis

Thuja occidentalis

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Cupressaceae

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Tree

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Arbor Vitae 

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             The Od Force of the leafy branches is used as one of the components in the preparation of the remedy. As herbal it is non-poisonous.

             The Od Force sweeps the dangerous toxic attributes of the GABA (gamma aminobutyric acid) receptor antagonistic. In this capacity it reverses the sufferings of asthma attack, intestinal irritation, excess stimulation of the nervous system and spontaneous abortions (miscarriage). It withdraws the spasm, caused seizures as well as the damages of the liver and the kidneys. It cancels the poisoning. In respect of the skin or eye it lifts the severe irritation and burns. It cures the rashes, skin diseases, warts if it is within its capacity. People with seizure disorders or gastrointestinal problems like ulcers or gastritis welcomes this force unknowingly.

It lifts the diversions caused from the chemically treated bronchitis, skin infections, cold sores and cancer. The diversion caused from the chemically treated pains of the osteoarthritis, joints and muscles. It lifts the focus newly appeared due to chemically ordered the nerve disorder of the trigeminal neuralgia affecting the face as this made order is caused at the cost of loss of further loss from the remaining fund of the Od Force of the suffering human system. Immunostimulant boosting over the immune system to loose the phlegm as an expectorant is withdrawn by this Od Force. It erases the diversions caused from brain problems.

It is peculiarly equipped to stop the queasiness, vomiting, painful diarrhea to death.

If the immune system is made too active then this activation may increase the symptoms of autoimmune diseases. And such diseases of multiple sclerosis (MS), lupus (systemic lupus erythematosus, SLE), rheumatoid arthritis (RA) and other conditions are the subjects of this Od Force. It also bats for some serious side effects caused chemically. The diversion caused further in the tackling or in decreasing the toxic effects of chemotherapy and radiation therapy is withdrawn as this Od Force is administered. The chemical treatment of the throat and the respiratory distress gunned to the congestive heart failure is vanquished in the use of this Od Force. The damaged done due to chemically diuretic increase of urination and chemical astringency applied to purify the blood, to reduce inflammation to cleanse the body of toxins cause harm in some new zone of the human system as these chemicals function only at the cost of the further subtraction of the previous fund of the pretreated condition and naturally this Od Force in its capacity lifts the system from the depth of additional loss of the Of Force.

In the process of inhibiting metastasis of tumor cells cell-mediated immune system is stimulated and pro-inflammatory cytokines is decreased. The Od Force in question to reverse the diversion caused from the said inhibition in the result of the said stimulation of the cells cell-mediated immune system and pro-inflammatory cytokines decrease, vibrates the cell to cell electromagnetic network of the said suffering human system that adds the deficient Od Force which had been lost during the process of the said inhibition and hereby the pre-inhibition condition is reched. In this reversing process all of the cytotoxic T-lymphocyte (CTL) activity, natural killer (NK) cell activity, antibody-dependent cell-mediated cytotoxicity (ADCC) and antibody-dependent complement-mediated cytotoxicity (ACC), pro-inflammatory cytokines and tissue inhibitor matrix metalloproteinases (TIMP) walk back. Enhanced the NK cell activity, ADCC and ACC much earlier than the control tumor, are withdrawn. It causes increase in the decreased elevated level of pro-inflammatory cytokines such as interleukin (IL)-1β, IL-6, GM-CSF and tumor necrosis factor (TNF)-α. The elevated level of antitumor factors such as IL-2 and TIMP are alco decreased. Thus previous immune surveillance is reestablished.

Mitogenic and cluster-forming activity causes T cell induction, in particular, of CD 4-positive T-helper/inducer cells as opposed to B cells. The CD-4+ T-helper/inducer cell induction is connected to an increased production of IL-2. The Od Force in question here reverses the total process if it is in its capacity. It may lift the induction over the CD4+ fraction of the peripheral blood T-cell subset. It is a potent exhibitor of the inhibited expression of HIV-1-specific antigens and HIV-1-specific reverse transcriptase. It may reverse the cytokine pattern induced in peripheral blood lymphocytes (PBL) and monocyte /macrophage cultures. It may withdraw the induction over IL-1 beta, IL-2, IL-3, IL-6, gumma-IFN, G-CSF, GM-CSF and TNF-beta production in PBL cultures and IL-1 beta, IL-6 in monocyte /macrophage cultures.

GABA is a chemical messenger that is widely distributed in the brain. Its natural function is to reduce the activity of the neurons to which it binds.It is the brain’s main inhibitory neurotransmitter. Some medications can enhance the natural reducing neural effect of GABA. It is estimated that close to 40% of the synapses in the brain work with GABA and therefore have GABA receptors. GABA receptors are channel receptors. This means that when GABA binds to them, they change shape slightly to allow ions to pass through their central channel. This channel mainly allows negatively charged chloride ions to enter the neuron to reduce its excitability. Because of this property of the GABA channel receptor, GABA is classified as an inhibitory neurotransmitter, as opposed to excitatory neurotransmitters, such as glutamate which augment the nerve impulses in the neuron. Glutamate receptors are responsible for the glutamate-mediated postsynaptic excitatation and are important for neural communication, memory formation, learning and regulation. GABA is the natural “key” to the GABA channel receptor’s “lock”. But GABA is not the only molecule that can modify this channel receptor’s opening. Other molecules can also affect it.

Naurally the Od Force here withdraws the diversion caused due to inhibition functioned by the GABA transmitters. Not only that, it makes free from its bondage with the other molecule or other medication to exclude those to escort the human system in its said pre-inhibitory stage. Erasing the faulty connection of GABA with the Glutamate receptors it may back the sufferers the usual neural communication, memory formation, learning and regulation. Again during the inhibitory period the GABA’s bondage with neurons causes change of shape slightly to allow ions to pass through their central channel. The Od Force in question reverses this process from inward to outward.

At least two distinct classes of GABA receptor, GABA_A and GABA_B are there. They differ in their pharmacological, electrophysiological and biochemical properties. Electrophysiologically the GABA_A-receptor complex mediates an increase in membrane conductance with an equilibrium potential near the resting level of −70 mV. This conductance increase often is accompanied by a membrane hyperpolarization, resulting in an increase in the firing threshold and, consequently, a reduction in the probability of action potential initiation, causing neuronal inhibition. This reduction in membrane resistance is accomplished by the GABA-dependent facilitation of Cl⁻ ion influx through a receptor-associated channel. On the other hand, increased Cl⁻ permeability can depolarize the target cell under some conditions of high intracellular Cl⁻. This in turn potentially can excite the cell to fire or to activate Ca²⁺ entry via voltage-gated channels and it may be a physiologically relevant event, especially in embryonic neurons. Electrophysiologically there remain two GABA-recognition sites per GABA_A-receptor complex. An increase in the concentration of GABA results in an increase in the mean channel open time due to opening of doubly liganded receptor forms, which exhibit open states of long duration. The increase in the ionic permeability of the GABA_A receptor complex is transient in the continuing presence of agonist. This phenomenon is known as desensitization and is rapidly reversible GABA_B receptors are coupled indirectly to K⁺ channels. When activated, these receptors can decrease Ca²⁺ conductance and inhibit cAMP production via intracellular mechanisms mediated by G proteins. GABA_B receptors can mediate both postsynaptic and presynaptic inhibition. Presynaptic inhibition may occur as a result of GABA_B receptors on nerve terminals causing a decrease in the influx of Ca²⁺, thereby reducing the release of neurotransmitters.

If the above narrated activities are once established the human system becomes escorted on a new disease focus shifting from the focus of the the previous disease as the said establishment comes in addition with the the already sufferings of the human system as diversion.The said establishment undergoes a reversed tract travel under the suviellence of the Od Force in question here to back to the previous condition of the diseased human system which results the withdrawal of the inhibition.

The complex includes five major binding domains. These include binding sites localized in or near the Cl⁻ channel for GABA.  These binding domains modulate receptor response to GABA stimulation. All establishments undergo the withdrawl of the said burdens of the bondages as the said Od Force activates the deactivation/stimulation caused due to binding. The benzodiazepine receptor is an integral part of the GABA_A receptor—Cl⁻ channel complex. Benzodiazepine receptor-binding sites copurify with the GABA-binding sites. The benzodiazepine agonists enhance GABAergic transmission.The frequency of channel opening in response to GABA beomes increased. Bonding at the binding sites may act by increasing the proportion of channels opening to the longest open state while reducing the proportion opening to the shorter open states resulting in an overall increase in mean channel open time and Cl⁻ flux. Channel blockers cause a decrease in mean channel open time working by preferentially shifting opening channels to the briefest open state. Both appear to act on the gating process of the GABA_A receptor channel, but their effects on the open states are opposite to each other. An action may cause preventing Cl⁻ channel permeability. The channel blocker with a net negative charge, blocks the channel by interacting with the positively charged amino acid residues within the channel pore, consequently occluding Cl⁻ passage through the channel. The Od Force lifts all the said blocks and inhibition to normalize the conditions.

Actually the Od Force in question withdraws the diversion caused by the enhanced GABA-mediated Cl⁻ conductance. It finally cancels the strong positive relation that was played chemically to cause stimulation of GABA-mediated Cl⁻ uptake, amino acids critical sensitivity in the membrane-spanning domains etc.

Intoxication activities like vomiting, stomach ache, diarrhea and gastroenteritis follow the absorption disorders, headache, nervous agitation and chronic convulsions, and symptoms of liver and renal toxicity extending to yellow liver atrophy, arrhythmia and myocardial bleeding is withdrawn by this Od Force. This Od Force is highly appreciated in withdrawing the induced severe metabolic disturbances. Intoxication may be accompanied by an irritant effect on the gastrointestinal tract, uterus, liver and kidney which may also be withdrawn in the activities of the Od Force.

The development of immunopharmacological potential over the human system is highly liftable from its sitting in the human system.   It is highly capable of erasing the inhibited human immunonodeficiency virus (HIV)-dependent cell death. MT-4 cells remain unaffected.  Whereas infected MT-2 cells are under the control of this Od Force to exhibit the inhibited HIV-1-specific antigen expression.  

 It causes withdrawal of the stimulation to increase in the proliferation rates of spleen cells. It is highly anti-mitogenic on the peripheral blood leukocytes.It is easily learnable that caused T-cell induction particularly of the CD4-positive T-helper/inducer cells in connection with an increased production of interleukin-2 (IL-2) may be vanquished. The mitogenic and cluster-forming activity causing T-cell induction particularly of the CD4-positive T-helper/inducer cells in connection with an increased production of interleukin-2 (IL-2) may be withdrawalable easily. This indicates that not only proliferation but also a differentiation to fully functional T-helper cells taken place is withdrawable by this Od Force. Only T cells, not B cells, come under the stimulation of this Force. The activation occurrs only in the presence of the monocyte/macrophage fraction and it comes under the neutralization by antibodies against interferon (IFN)-γ and IL-1. Induced IFN-γ production in CD4⁺ cells was assumed to be the mode of action, whereby the IFN-γ produced stimulated monocytes/macrophages to produce IL-1 is also under reversible under this Od Force. As a second signal of the T-cell activation, this triggeres the expression of IL-2 receptors and the production of IL-2 in CD4⁺ T-helper cells. In all these cases impending actions may be taken back by this Od Force. Any deviation may be backed by this Od Force.

The increasing secretion of the cytokines IL-1, IL-6 and tumor necrosis factor-α (TNF-α) is also controlble in this Force. . It is capable of reversing the reversed process that may increase in the production of IFN α_β. 

The Od Force in question causes influence to impose anti-oxidative measure on the oxidative metabolisms of the macrophases. It causes a decrease in the number of antibody-producing lymphocytes in the hemolytic plaque assay. 

Steffensia elongate

Steffensia elongate

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Piperaceae

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Shrub

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Matico

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The Od Force of dried leaves is used as one of the components in the preparation of the remedy. It is non-poisonous.

The Od Force is qualified with the presence of a wide range of the Electrohomoeopathically active faculties of energy obtained from the plant including those of flavonoids, sequiterpenes, monoterpenes, heterocycles, phenylpropanoids, alkalods and benzenoids etc. It lifts the diversion caused from styptic activities. Addition causing diversion from the chemically stopped bleeding from the lungs, stomach and kidneys, also from intestine in the form of dysentery is also a subject of the withdrawing qualities of this Od Force. The diversion due to chemically dispersed congestion causing leucorrhoea, gonorrhoea, catarrh of the bladder, piles and chronic mucous discharges is also erased by this Od Force if it is in its own capacity. Similarly the diversion caused from the chemically treated dyspepsia backed by the mucus affections of the stomach lashed from behind by the blood congestion is also under this Od Force to lift the same diversion. Shift due to addition caused from the chemically arresting haemmorages from the wounds leech bites etc. may also be withdrawn by the use of this Od Force. Diversion resulted from the chemically obtained easement from the gastric disorders, tumours and cuteneous diseases may also be lifted by this Od Force.

An exertion of a vital action on bleeding vessels is necessary so as speedily to arrest the haemorrhage. It may also be necessary to check other discharges, such as the profuse expectoration and also the night-sweats of consumptive patients. Due to some loss of the Od Force of the said human system the there was causing the bleeding, other discharges. At this situation if a chemical exerts some action on the bleeding vessel to stop the said haemorrhage or discharges then another chemical reaction will be caused in the said human system and for this reason another bulk of the Od Force will be lost. As a total the suffering will be vanished to give a new suffering which is/are not the previous. The Od Force is capable of erasing the loss of the Od Force that was caused to chemical activities to stop the bleeding or other discharges to escort the diseased system in its previous disease status. Diversions caused from the chemically treated genitor-urinary complaints, atonic diarrhoea and extraction of teeth.

It withdraws the diversion caused from the cytotoxic activities, also from the antimicrobial functions of the selected chemicals over the human physique before the control of the same. It is also effective against the added sufferings caused by the chemical reaction when the gynecological maladies and vaginitis. Diversion caused by the stanched blood is excellently taken away by this Od Force. It is also excellent to lift the diversion caused from the arrest of the venous haemorrhages. Its efficacy is highly noticeable in erasing the diversion obtained from the chemically treated nasal catarrh. It is highly capable of taking up with the damages done by the Syphilis before being expulsed from the human physique.

It imposes its activities in repairing the damages done by the bacteriostatic and fungistatic footings of Trichophyton, mentagrophytes, pseudomonas aeruginosa, candida albicans, Cryptococcus neoformans, aspergillus flavus, aspergillus fumigatus, Escherichia coli.

The diversions caused from the chemically treated digestive disorders including stomach aches, vomiting, gastric ulcers, intestinal gas and stomach cancer, urinary tract infection, cystitis, venereal diseases such as gonorrhoea and trichomonas.

Taken as an infusion, the leaves are widely used as a remedy for all types of digestive disordluers incding stomach aches, vomiting, dyspepsia, diarrhoea, gastric ulcers, intestinal gas and even stomach cancer; they are considered an excellent genitourinary tonic and are used in the treatment of kidney stones, urinary tract infections, cystitis, urethritis, leucorrhoea, vaginitis, and various venereal diseases such as gonorrhoea and trichomonas; they are also employed for various upper respiratory conditions such as bronchitis, pulmonary haemo accidents.

Solanum dulcamara

Solanum dulcamara

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Solanaceae

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Shrub

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Bitter sweet

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The Od Force of the leaves and young branches is used as one of the components in the preparation of the remedy. As herbal it is narcotic. It is poisonous.

The tumor-inhibitory activities against cancer like sarcoma, warts etc. generally leads to focus differently as the said chemical activity reaction with the selected chemicals of the said suffering human system causes further loss of the Od Force. This Od Force lifts this additional loss, if it is within its capacity. Its battle against the toxicity and teratogencity has made it qualified to appear as the component of the remedy Ven1 basically.

In performing its duties of fighting against the diversion caused from the treated cancer chemically it appears to impose cancellation over the functions posted in the suffering human system by the biological immune response modifier.

 In this process of cancellation it cures the caused renal congestion, occasionally augmented urinary secretion of an albuminous nature, exerted depressing or paralyzing effects upon the respiratory nervous system, caused increased but enfeebled cardiac action, tetanic spasms of the thoracic muscles as well as those of the extremities and thereby increased sensitiveness of the cutanious nervous system. Thus indirect influence upon the brain, stomach or bowels is withdrawn. It sweeps the diversion focused on the spinal chord and medulla oblongata caused due to chemical treatments of the pulmonary maladies attended with the spasm or irritation.

It adds the lost Od Force caused from the chemical treatment of skin diseases, warts, tumours, felons etc. The chemical activities that has caused the paralyze of the central nervous system, slowing of the heart and respiration, lowering of the temperature, causing vertigo, delirium, convulsions and death is come to a cancellation if this Od force takes the charge of vibrating of the cell to cell Electromagnetic field of the said diseased human system that has been made deactivated in the process of the chemical activities. It erases the diversion caused from the chemically treated diseases like alterative, anodyne, depurative, mildly diuretic, emetic, expectorant, hepatic, mildly narcotic and purgative[

It is also used in the diversions caused from the chemically treatment of arthritis, rheumatism, bronchial congestion, heart ailments, ulcerative colitis and jaundice, cellulites etc. This is also used in tackling to cause an end of the new focuses of the variety of complaints including backaches, cough, diarrhoea, eye inflammations and joint pains, eczema, furuncles (boils), acne, warts, cancerous sores and other swellings chemically treated.

Its main usage is for regaining conditions that had an impact on the skin, mucus membrane and the membrane (synovial membrane) around the joints before they have been treated chemically.

Caused hemolytic and hemorrhagic damage to the GI tract without any confusion with bacterial gastroenteritis, with a documentation of weak effect on cardiovascular function is under the control of this Od Force.

Caused renal congestion, and occasionally augmented urinary secretion of an albuminous nature exerts a depressing or paralyzing influence upon the respiratory nervous system, causes increased but enfeebled cardiac action, tetanic spasms of the thoracic muscles as well as those of the extremities, and increases the sensitiveness of the cutaneous nervous system. The said withdrawal Od Force lifts the heart and the arteries etc. from their depressed states.

Circulatory and respiratory depression, convulsions, cyanosis, death, diarrhea, dilated pupils, headache, paralysis, scratchy throat, shock, speech difficulties, stomachache, subnormal temperature, vertigo, and vomiting etc are the victims of this Od Force. The pathologic changes in the GI tract (glandular mucosal necrosis and necrosis of the small intestine) are also under the supervision of this Od Force to repair the same.

The Od Force in question withdraws the diversion caused from the chemically removed obstructions in the liver and gall bladder and cured spleen problems and jaundice and also the diversion caused from the speedier removal of the congealed blood on the surface or within the body cavity and even speedier healed beaten or bruised chemically. It erases the additional diversion due to increased or influenced waste and excretion, marked influence on the cerebra-spinal centers, normalized secretions after exposure to cold and damp chemically. This Od Force is needed to cancel the loss of the Od Forces in the material treatment of syphilitic disease, rheumatic disease, cachectic affections, ill conditioned ulcers, tuberculosis, scrofula, scrofulous cachexia, indurations from milk, jaundice, inflammatory deposits, chronic diseases in which circulation is feeble, fullness of tissues and tendency to oedema, gouty conditions, diseases characterized by impairment of the blood, rheumatism or secretion abnormalities. Cardiovascularity with impaired circulation is also under the consideration of this Od Force. Diversion resulted from the chemically treated rheumatism or secretion abnormalities which had been resulted from long continued exposure to cold and dampness, eruptive fevers, obstructed menstruation, leucorrhea, suppression of the menses, with headache, nausea is also erased.

It saves the human system from the curse of the super diversion caused in the disguise as innocuous and causing minimal disorders in the physiological functions originated from the Adaptogen activities. In this run it even escorts the human system from the hazards of the caused vomiting, vertigo, convulsion, feeble heart and paralysis.

A chemical may increase resistance to viral infection like herpes, polio. A chemical may increase resistance to bacterial infection. A chemical may increase resistance to tumours of bladder, breast and cervix. A chemical may increase resistance to liver damages. A chemical may increase resistance to cancer, free radical damages. A chemical may increase resistance against a poisoning. A chemical may increase resistance to viral infection. In fact in every case as mentioned above the focus that receives the above mentioned troubles comes to a new focus after chemical reaction between the human system and the chemical used and at this new focus the above suffering does not get a link with human system . So the resistances as stated do not get a link with the human system. Even a single chemical may cause all of the focuses as stated shifted. So the said single chemical being single can put a resistance against above stated all in single new focus. Due this new single focus any of the above stated suffering can not throw its anchor in the human system. But this incidence happens with loss of the Od Force for each separate case. Naturally the single chemical that takes the total responsibility of all of the hazards at the same causes a bigger loss of the Od Force from the already remained Od Force of the said human system. The Od Force sweeps the diversion causes due to the total loss singly smartly if it is within its capacity.

The chemical that normalizes the changes associated with stage of exhaustion including mucous membrane and skin ulceration, abnormal mucous membrane secretion, catabolic shifts in metabolism, wasting, weight loss, abnormalities in capillary circulation, membrane permeability abnormalities, endocrine abnormalities, temperature abnormalities, and dyspeptic states of the digestive tract writes a story of such loss of the Od Force of the human system to escort the suffering human system on a new focus backed by an additive loss to the already remained Od Force of the said human system alien to the link that the above sufferings clicks to establish their existence in the human system. The compounds that have been shown to normalize inflammation, hypercholesterolemia, ulceration, bronchoconstriction, temperature abnormalities, and rheumatism act the same script only with different amount, different magnitude etc.

Rosa canina

Rosa canina

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Rosaceae

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Shrub

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Dog Rose

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The Od Force of the ripe fruits with seeds is used as one of the components in the preparation of the remedies. It is non-toxic generally.

The Od Force in question silently withdraws the underlying worsening causes behind the increasing spread of cancer without having any effect on the cancer cell growth, especially in case of prostate cancer.

It sweeps the danger resulting from the halted or reversing in the growth of cancer or reducing the incidence of cancer.

It reverses the focus caused as anti-diabetic effects and antioxidant potential. It has involved itself greatly in case of withdrawing the diversion resulted from the process of expulsing stone and gravel. In order to prevent the damage to fragile capillaries some chemical complex may strengthen the body tissues and may help to build and maintain a healthy vascular system, but this is at the cost of the deactivating the electromagnetic field, vibrations or undulation of which causes the material existence of the human system. Naturally this deactivation causes due to loss of the Od Force that would maintain those zones of tissues as because in this process of strengthening there needs the loss of the Od Force for taking part in the chemical reaction takes place in the said process of strengthening body tissues, building and maintaining forcefully the health of the vascular system.

In patients suffering from osteoarthritis, rheumatoid arthritis, and low back pain etc. the lipophilic constituents may involves themselves in the mechanisms of actions of causing anti-oxidative and anti-inflammatory effects. In its capacity the issue of the diversions done by the aforesaid mechanisms here may be reversed by this Od Force in question. For these reasons it is so important to consider the usefulness of this Od Force to pay the debt of the costing of probiotic, stool regulating and the smooth muscle-relaxing actions as well as lipid-lowering, antiobese and anti-ulcerogenic effects. It bounces back the effects caused from the chemically treated skin diseases.

It affects the tissue regeneration properties. It is value in erasing the damage happened to open new focus of suffering in the process of reducing scar tissue and stretch marks caused by pregnancy and birthing.

It holds the unusual laxative condition in the digestive organ. It cures the irritation in the mouth as well as the digestive tract. In the same principle it is anti-diuretic. It withdraws the boosting activities over the immune system that were used to protect infections or to curtail the sufferings of the infections of colds, flue etc.

It withdraws the gastrointestinal toxicity. It helps to escort the human system from the damages done by the ultraviolet B radiation. Free radicals associate with pathogenesis of various disorders and diseases such cancer, cardiovascular disease, osteoporosis, diabetes and cataracts. Again chemically the antioxidant enzymes superoxide dismutase (SOD), glutathione peroxidase (GSH-Px), glutathione reductase (GR) may be restored, glutathione (GSH) may be reduced, level of the lipid peroxide malondialdehyde ( MDA) may be decreased in hypertensive patients, A favourable chemical effect may reduce MDA levels and increase GSH levels in coronary artery disease in postmenopausal women. In reality the above radicals express the deficiencies of the Od Force for which there appear the cancers. Similarly the deficiencies may be expressed in the said hypertension and coronary artery diseases. The chemical is used to cut off the above pathogenesis due to its chemical reaction with human cell system will cause another deficiency to divert the focus of sufferings to cope the sum of the two sided deficiencies. So in order to knot of the new sufferings of new focus the Od Force here is very important to be taken into consideration.

It has great effect on the lipid micelles in the small intestine mainly. It withdraws any diversion caused in the processed of formation of hydrophilic form. Not only that, it lifts the permeated activities in the intestinal mucosal cells by an active mechanism. It decreases the unauthorized load of the lipoprotein fractions in the blood plasma. It disperses the unusual gatherings of lipoprotein fractions in fatty tissues and organs such as the adrenal glands, liver and testes to eliminate those from the human system.

It exhibits the essayed effects from inhibited iron-catalyzed lipid peroxidation and nitric oxide production. It is also capable of reversing the damages happened due to activity of inflicting tissue damages done by the reactive species like peroxynitrite (ONOO–). In order to protect the stress-induced DNA damage there may cause the scavenging of intracellular reactive oxygen and/or nitrogen species at the cost of opening of new focus to produce more dangerous sufferings and to lift the same this Od Force in its capacity is extremely helpful. All of the decrease in glucose levels, an increase in insulin concentration, a decrease in H2O2 and thiobarbituric acid reactive substances levels, increased total antioxidant status, and increased antioxidant enzyme activities (ie, catalase, superoxide dismutase, glutathione peroxidase) with improvement in serum lipid profile in respect of diabetes are not some easy incidences. All are at the cost of doing damage in some different field of sufferings making symptomatic relieves. The Od Force that was controlling the said human system comes to more serious deficiency in its fund to cause more grave sufferings. It also lifts the diversion caused from the treated diabetic retinopathy chemically.

Inflammatory mediators such as tumor necrosis factor (TNF-α), interleukin (IL)-1β, and IL-8 enhance binding of low-density lipoprotein to endothelium and up-regulate expression of leukocyte adhesion molecules on endothelium during the process of atherogenesis. In this situation inhibition of TNF-α-induced NF-κB activation, ICAM-1 expression, and monocyte-endothelial interaction in human umbilical endothelial cells may be caused chemically, FurtherTNF-α-induced IκB phosphorylation, NF-κB expression, and NF-κB p65 translocation from cytosol to nucleus may also happen, TNF-α production may be restricted are also at the cost of damages of the inner sense. We may get such at the cost of exhibition of the antiatherogenic effects by inhibiting the expression of inflammatory mediators in hyperhomocysteinemic conditions. This Od Force is quite sound in reversing the same as if imposing the inhibitory effect on the exhibited antiatherogenic effects by exhibiting the expression of inflammatory mediators in hyperhomocysteinemic conditions. We may notice significant linkage between the inhibition of edema, induced ischaemia-reperfusion and liver injury, the injury being the new focus. The chemical barrier against the oxidative DNA damage in leukocytes and prostate tissue may lead the human system to some much grave situation. To escort the same from the danger to previous state the use of the Od Force here is urgent. We may use some selected chemical to increase the preservation of the myocardial antioxidant status and alteration of the haemodynamic parameters to protect the occurrence of the myocardial injury after ischemia and reperfusion but at the cost of the happening of the incidence of the new focus. To erase this focus this Od Force may be needed if it is in its capacity.

Liver damage is associated with cellular necrosis, increase in tissue lipid peroxidation, and depletion of tissue GSH levels. In addition, serum levels of many biochemical markers like serum glutamic oxaloacetic transaminase, serum glutamic pyruvic transaminase, triglycerides, cholesterol, bilirubin, and alkaline phosphatase are elevated when liver damage is present. Chemically this crisis may be managed by applying the hepatoprotective activity on the process of the damaging of the liver at the cost of reducing the levels of cholesterol, triglycerides, and free fatty acids, followed by a decrease in the levels of phospholipids in the serum and the liver. Chemically antioxidant liver enzymes, such as glutathione peroxidase, glutathione-s-transferase may be restored to protect the gastric carcinogenesis. But all of the above at the cost of serious breakdown expressed in some other field/place of the human system as a focus to register the sum of the already lost Od Force and newly lost Od Force due to chemically triggered vanishing of the lying symptomatic sufferings. The Od Force in question is finally adamant to lift the dangers caused by this chemical activity against this gastric carcinogenesis, even hepatocellular carcinoma remaining in its capacity.

 Diversions caused by the process of preventing or treating to reduce the cancers of the pancreas, colon and rectum, esophagus, oral cavity, breast, and cervix is also a subject of this Od Force to erase the same. The reduction of the tumor metastatis is subjected to the action of attenuation of tumor invasion, proliferation and angiogenesis. In solving the problem further coiled with such attenuation of tumor invasion, proliferation and angiogenesis the human system needs this Od Force. This may be solved by the exhibition of the inhibited growth of human colon cancer HT-29 cells causing reversed action on the associated down-regulation of the PI-3K/Akt/mTOR signaling pathway. This Od Force is equally expert in exhibiting the inhibited platelet-derived growth factor-BB–induced signaling and cell migration in human cultured skin fibroblasts through a direct unbinding to platelet-derived growth factor-BB.

The antiproliferative and apoptotic activities on various cell lines, such as human colon carcinoma (HuCC), B chronic lymphocytic leukemia (EHEB), human erythroleukemia (K562), and Raji, a prototype of Burkitt lymphoma cell line become bound to face the opposite actions. And this Od Force does the same. It reverses the inversed association with developing prostate cancer to lift the diversion caused by the respective chemical. It withdraws the diversion activity workable on the androgen-sensitive (LNCaP) and androgen-independent (PC3 and VeCaP) prostate cancer cell lines after the issuance of the chemical induction of apoptosis and inhibition of cell growth. In cases of diversions caused from chemically treated both of hormone-refractory and hormone-sensitive prostate cancer it is also equally excellent. It also exhibits the inhibited progression of benign prostate hyperplasia. In this process of withdrawing the diversion it cancels the interference with growth factor receptor signaling and cell cycle progression, specifically in prostate cancer cells. The up- regulation of the expression of the gene connexxin-43 causes the direct intercellular gap junction communication (GJC). GJC is deficient in many human tumors, and its restoration or up-regulation is associated with decreased proliferation. So after withdrawing the diversion effect imposed on this communication expression by this Od Force we should search for that deficiency in the total fund of the Od Force of the human system the supply of which will protect the fall of the GJC. The Od Force has connected itself with the diversion resulted from the inhibition of the breast cancers.

Cataracts are multifactorial disease. Osmotic stress with weakened antioxidant defense mechanisms is attributed to the changes observed in diabetic cataract. Nutritional anti-oxidants slow down the progress of cataract and age-related macular degeneration. If the ARPE-19 the retinal pigment epithelium cell line is activated chemically to protect H2O2-induced oxidative stress it will affect the human system through some new focus. So to erase this focus this Od Force may be needed. It also withdraws the stress caused by the chemically decreased serum and lipoproteins in age related macular degeneration. It refunds the loss of Od Force caused by sugar-induced morphological changes and modulated antioxidant status of lens epithelial cells, galactose-induced cataract. In case of chemically protected male infertility it sweeps the stress over the process that remains engaged in producing the unusual excessive ROS-containing free-oxygen radicals functioning against the redox defense that mechanism that acted against the infertility. Naturally the deactivated portion of the framework of the human cell to cell and gene to gene electromagnetic network to normalize the infertility condition should be vibrated. It has significant role to erase the sufferings caused from the chemically improved sperm concentration and motility of the men with infertility.

In tackling the problems of osteoporosis chemically an action is made over the oxidative stress induced by ROS to hold up the same. A chemical stress is placed on proliferation and differentiation of osteoblasts (osteoblast-like osteosarcoma SaOS-2 cells), the cells responsible for bone formation. This stress adds another shortage of fund with shortage of the fund of the Od Foce for which the said proliferation is caused. In order to come out from the diversion caused from the stress put on the proliferation, differentiation of osteoblasts the Od Force here puts its functions to uplift the decreased level of NTx and also protein oxidation. Carbonyl levels, which are the product of protein oxidation, lead to oxidative stress and osteoporosis. So this Od Force plays for the increasing the levels of carbonyl to make a pavement towards the allotment of another fund of the Od Force that will act to erase the causation of the proliferation and the differentiation.