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Betulaceae
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Tree
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Silver Birch
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The Od Force
of the branches, bark and leaves is used as one of the components in the
preparation of the remedy. It is non-poisonous.
To lift the
potentiality of the dermal irritation it acts anti-astringent. The potentiality
that applies itself to put its purifying properties to heal the skin through
the process of facilitated excretion of the fluids, promoted metabolic activity
with a leave of irritation with a grave base, in the skin is withdrawn
successfully. Anti-inflammatory and skin soothing chemical
abilities, as are used in the treatment of eczema, psoriasis and warts causes a
diversion ending into some serious troubles to open a new focus of the
sufferings caused from the sum of the previous sufferings plus the new by this
Anti-inflammatory and astringent activities.
It acts as anti-laxative to back the intestine
in normal laxative status. It is anti-diuretic. It withdraws the diversion
effects caused from the chemically treated gout, rheumatism, arthritis,
nephritis etc. It also erases the damage done by the chemical process imposed
over the kidney to make free the same from stones. The flush out of the water
from the urinary tract done chemically happens at the cost of affecting the
kidney, bladder, ureters and urethra. So in its works the Od Force put, removes
all the stress that come in the process of flushing out the water or expelling
the stones, treating the cystitis. So it rescues the human system from the
danger of lob blood pressure resulted from the flushing of the water
chemically.
It has
excellent effects on the allergy in its capacity. In order to low the high
blood pressure it intervenes in the unusual retaining of the sodium working
against the process of increasing intake of the amount of salt (sodium).
The antineoplastic
chemical treatment of the malignant melanoma gets its success at the cost of
the damage of the sensitive mitochondria of the spermatozoa. The Od Force in
question plays it important role in gaining the said sensation of the same by
putting an anti- induced activity in the mitochondrial pathway of apoptosis.
Human spermatozoa post-ejaculation shows all elements of intrinsic (via
mitochondria) and extrinsic (via death receptors) programmed cell death or
apoptosis. The Od Force here is completely sincere to reverse the cytotoxic
inducing direct effects over the intrinsic apoptosis caused on the
mitochondria. It reverses the activity that was laid on to cause an immediate
disruption on the mitochondrial transmembrane potential and activation of the
“death enzymes” caspase-9 and -3. This activity of this Od Force for protecting
the loss of the mitochondrial potential (mTMP) cuts the accompanied significant decrease of spermatozoal motility
if it in its own capacity.
Diversion
caused due to chemical activity that deals the prostate cancer may easily be
erased by this Od Force. In its function it exhibits the process that was
adopted in the Inhibition
of the ubiquitin-proteasome system (UPS) of protein degradation which is taken as a valid anti-cancer strategy
chemically. It exhibits the inhibited multiple deubiquitinases (DUBs), which
resulted in the accumulation of poly-ubiquitinated proteins without influencing
the normal fibroblasts. Exhibition of the inhibition of DUBs and induction of
apoptotic cell death specifically in prostate cancer but not in normal cells
and tissues provides us an effective measure in rescuing the human system from
being carried the severe threat in the genes to the generation after generation
to the off-springs.
The Od Force
is efficient to withdraw the diversions caused by the chemically treated
cancer. It is equally smart to stop and reverse the process that causes types
of tumor cells to start a process of self-destruction of apoptosis. It appears
fastened to sweep the slow process of diversion caused from the growth of the
tumour cells and the human of tumor cells and the human immunodefiency virus
(HIV). The cease of Diarrhea and dysentery may seriously be refocused into
gravity of cancer due to some short of deficiency added by the loss of the Od
Force of the existing human system. The process of increase of the
sensitivities of the cancer cells reverses through the causation of the
decrease of the said sensitivity in the withdrawal of the diversion. It lifts
the diversion issued through the killing of the melanoma. It erases the
sufferings appearing as new focus that peeps after the medicinally treated or
cured actinic keratosis precancerous cancerous skin condition. It has involved
itself with the diversions caused by the chemically treated some nervous tumour.
In neuroectodermal tumor cells the induced apoptosis
is accompanied by caspase activation, mitochondrial membrane alteration and DNA
fragmentation. Caspases are produced as inactive proyenzymes which are
proteolytically processed to their active forms. These proteases can cooperate
in proteilytic cascades, in which caspases activates themselves and each other.
The initiation of the caspases cascade may lead to the activation of endonucleases
such as caspase-activated DNAase (CAD). After activation, CAD contributes to
DNA degradation. Naturally in order to escort human system from the
additionally sufferings caused by the induced apoptosis this Od Force functions
exactly in opposite direction. It lifts the induced apoptosis that causes
direct effects on mitochondria, leading to cytochrome-c release which in turn
regulates the “downstream” caspase activation. The Od Force in question exerts
its exhibitory activities on the inhibitory effects on metastatic malenoma
partly by decreasing p53. It is also preferential if the same is within its
capacity, to withdraw the apoptotic effect on C8161 metaststic melanoma cells,
with greater DNA fragmentation and growth arrest and earlier loss of viability
than their nonmetastatic C8161/neo 6.3 counterpart. It is able to cause
withdrawal activities on induced two different cytotoxic and cytostatic effects
at the same time. The Od Force is selective for tumor cells those experience
minimal toxicity against normal cells. The effect of this Od Force on the
melanoma cell lines is stronger than its growth exhibitory effects on primary
melanocytes.
The diversions
caused, selective for lifting or withdrawal of are the following----
A.
Gastrointestinal
------------------------
1. Gastric mucosal lesions
2. Gastrointestinal
bleeding.
3. Anorectal ulceration and rectal stenosis
4. The risk of developing
dyspeptic events of epigastric pain,
heartburn, nausea, ulcers etc. low in
rheumatic patients without
having prior gastrointestinal symptoms .
5. Appendicitis.
6. More serious gastrointestinal effects
include hemorrhage, peptic
ulcers, perforation, small bowel
enteropathy, and esophageal
ulcerations.
B. Renal
-----------
Decrease in renal function, inhibition of
renal prostaglandin synthesis, decreases
in renal blood flow. Vasodilating renal prostaglandins in patients with arterial
underfilling (i.e. heart failure, cirrhosis).
Reduction in glomerular
filtration rate (particularly
in patients who are sodium restricted or who exhibit diminished effective
arterial blood volume, such as patients with advanced heart
failure or cirrhosis), interstitial nephritis, papillary necrosis, elevations
in serum creatinine, elevations in blood urea nitrogen, proteinuria, hematuria,
and renal failure.
C. Hematologic
------------------
Increased blood fibrinolytic activity. In
addition, hypoprothrombinemia, thrombocytopenia, thrombocyturia, megaloblastic
anemia, and pancytopenia rarely. Aplastic anemia and eosinophilia.
D. Hypersensitivity
---------------------
An up-regulation of the 5-lipoxygenase pathway
of arachidonic acid metabolism with
a resulting increase in the products of 5-lipoxygenase (such as leukotrienes).
Bronchospasm, rhinitis, conjunctivitis,
urticaria, angioedema, and anaphylaxis, bronchial asthma, and nasal polyps.
E. Dermatologic
-------------------
Stevens-Johnson syndrome and a lichenoid
eruption. Unilateral aquagenic wrinkling of
the palms and papuloerythroderm.
F. Hepatic
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Hepatotoxicity
and cholestatic hepatitis.
G. Oncologic
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Pancreatic cancer, the risk of large bowel neoplasms.
H. Metabolic
--------------
Dehydration and hyperkalemia. Respiratory
alkalosis and metabolic acidosis, hypoglycemia. displaced triiodothyronine (T3) and
thyroxine (T4) from protein binding sites. The initial effect is an increase in
serum free T4 concentrations.
I. Cardiovascular
-------------------
Migraine,
chest pain, tachycardia and orthopnea.variant angina, ventricular ectopy, conduction abnormalities,
and hypotension, congestive heart failure, intracerebral hemorrhage.
J. Nervous
system
----------------------
Agitation,
cerebral edema, coma, confusion, dizziness, headache, cranial hemorrhage,
lethargy and seizures. Tinnitus and subjective hearing loss (or both), impair
hearing performance, particularly in the setting of background noise
K. Other
-------
1. Reye's
syndrome typically involves vomiting, neurologic dysfunction, and hepatic
dysfunction, easy viral infections.
2. Musculoskeletal
Rhabdomyolysis
3. Respiratory
---------------
Hyperpnea, pulmonary edema, and tachypnea.
4. Endocrine
---------------
Hypoglycemia in children) and hyperglycemia.
5. Ocular
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Localized
periorbital edema
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