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Shrub
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Myrtle
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The Od Force of the leaves
and fresh flowering branches is used as one of the components in the
preparation of the remedy. As herbal it is poisonous.
This force is anti-diaphoretic,
anti-diuretic and anti-rubefacient. The Od Force is highly qualified to add
requisite Od Force to the human system soundly deficient for which the
respective genes have acquired of the mixed Constitutional sufferings
originated from the infections of Syphilis in the genes on the Mixed
Constitution we know in Electrohomoeopathy. In its capacity it lifts the
diversion caused from the chemically treated foul ulcers, rheumatisms and skin
eruptions. It subtracts the addition of loss of Od Force caused from chemical
antibacterial, skin-purifying activities. It redirects the diversions caused
from the chemical analgetic and mucolytic activities. In a group of components
in Electrohomoeo remedy it escorts the suffering human system from the load of
loss from the chemically treated bronchial diseases. Its effectiveness is to
redirect the dangerousness caused from the chemical efforts against the cancer.
It
lifts the diversion effects of the chemical activities behind the mitochondrial
apoptosis and senescence in Chronic Myelogenous Leukemia. It causes the reverse
of the process inducted for apoptosis in CML cells through down-regulation of
anti-apoptotic proteins and at the same it erases the induced senescence in CML
cells with a view of withdrawing the chemotherapeutical potential in the human
system.
In its activity it
reverses the direction of suppression along which the biosynthesis of eicosanoids causes. It is done by causing the exhibition over
the process of occurrence of the inhibition of 5-lipoxygenase and
cyclooxygenase-1 and to exhibit the inhibited release of elastase and the
formation of reactive oxygen species in activated polymorphonuclear leukocytes.
Here the selected Od Force of the plant expresses the typical proinflammatory
cellular responses. In this expression it may back edema and pleurisy lifting
the previously imposed or exerted anti-inflammatory effects in the pleurisy
chemically through a process of cancel of the activity that was remained
engaged in reducing the exudate volume and leukocyte numbers, lung injury and
neutrophil infiltration (myeloperoxidase activity), the lung intercellular adhesion
molecule-1 and P-selectin immunohistochemical localization, the cytokine levels
(tumor necrosis factor-α and interleukin-1β) in the pleural exudate and their
immunohistochemical localization in the lung, the leukotriene B4, but not prostaglandin E2, levels in the pleural exudates and lung
peroxidation and nitrotyrosine and poly (ADP-ribose) immunostaining.
Inflammatory diseases
are accompanied by neutrophil infiltration, release of proinflammatory
cytokines [such as tumor necrosis factor (TNF)-α and interleukin (IL)-1β] and
bioactive mediators [e.g., leukotrienes (LTs) and prostaglandins], and
expression of adhesion molecules [intercellular adhesion molecule (ICAM)-1 and
P-selectin]. This Od Force takes back the contribution to tissue damage
characteristic of the inflammatory process. In addition, it decreases the
elevation of intracellular Ca2+ concentration to stop the causation of
the release of proteases (e.g., leukocyte elastase or cathepsin G) and
formation of reactive oxygen species (ROS) both of which destroy invading
particles but damage cells and tissues of the host. Moreover, ROS generation
induced by intracellular Ca2+ elevation causes lipid peroxidation
and DNA single-strand damage. Interference with the generation or action of
these proinflammatory activities exerts detrimental effects in a variety of
inflammation finally as this makes the suffering human system steals of more Od
Force from the remaining fund of the Od Force of the same. It may be reminded
here that prostaglandin (PG)E2 is a prominent product of the cyclooxygenase
(COX) pathway and has long been considered as a mediator of inflammation, pain,
fever and cancer, but it is also known to regulate physiological functions in
the gastrointestinal tract, the kidney and in the immune and nervous systems.
It exhibits the inhibited mPGES-1. Moreover this Od Force advantageously lifts
the blocks over the LT formation and expresses the suppressed both of the LT
and the PG biosynthetic pathway to take the advantage of withdrawing the
inhibition of a single pathway, not only in terms of the inflammatory
effectiveness but also with escorting the human system suffering from the
gastro-intestinal problems to a cure condition. In regard of its effectiveness
in the whole of blood and its functionality on mPGES-1 this Od Force has a
definite activity to rub off the interesting pharmacological performances of
the dual mPGES-1 and 5-lipoxygenase inhibition. In respect of inhibition of the
COX enzymes product formation it may be stated that in its capacity if there is
suppression or inhibition the Od Force in question is quite skilled to lift the
same. It lifts the diversion caused by the suppression over the cellular COX-1
product formation in intact platelets. In case of COX-2 suppression the genetic
electromagnetic recordings may be so that pathological or pathohistological
supports we may not get, if it be so this Od Force is similarly smart to erase
the same.
The
Od Force involves itself in returning the human system its previous status in
respect of its medical sufferings from the new status obtained from the
chemically treated diarrhoea, peptic ulcers, hemorrhoids, inflammation,
pulmonary and skin diseases. In reality it possesses a broader spectrum of anti-pharmaceutical
and anti-therapeutical activities to lift the imposed chemical functions
anti-oxidative, anticancer, anti-diabetic, antiviral, antibacterial,
antifungal, hepatoprotective and neuroprotective.
The
Od Force withdraws the diversion caused due to inhibition of the breast cancer
cell line, prostate cancer cell line. The most sincere withdrawal activity is over
the diversion issued by the vanishing of the fibroblast cell line. In its
capacity it reverses the induced apoptosis in cancer line with marginal
cytotoxicity for non-transformed cells, via the mitochondrial cytochrome
c/Apaf-1/ Caspase-9. Cell death generally causes here via apoptosis and it will
be found it to be much less cytotoxic for non-transformed peripheral blood
mononuclear cells (PBMC) or foreskin fibroblasts, causation of processing of
poly (ADP-ribose) polymerase (PARP), caspase-3, -8, and -9 in PBMC. The Od
Force in question here reverses the induced apoptosis mediated by the intrinsic
rather than the extrinsic death pathway. So it lifts the impending loss of the
mitochondrial membrane potential in MM6 cells and revokes the evoked process of
releasing of the cytochrome c from mitichondria. Furthermore, Jurkat cells
deficient in caspase-9 resistant to the induced cell death is reversed in its
capacity. The anti-processing of poly (ADP-ribose) polymerase (PARP), caspase-3,
-8, and -9 in PBMC may be reestablished. In cell lines deficient in either CD95
(Fas, APO-1) signaling, FADD or Caspase-8 the potentiality to induce cell death
and PARP cleavage is withdrawn by this Od Force.
The
Od Force is capable of repairing effectively the damage done due to diversions
caused by the inhibition of aryl hydrocarbon hydroxylase (AHH) activity and
3H-benzo(a) pyrene (3H-BP) binding with the microsomal protein together having
back up inspiration of the chemical effects of the ethanol.
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